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Genetics Laboratory of Development and Diseases, Institute of Biotechnology, Beijing, P.R. China; and 2National Center of Biomedical Analysis, Beijing, P.R. China Hepatocellular carcinoma HCC ; is one of the most prevalent malignancies in the world. Epidemical investigations show that the infection of hepatitis B virus HBV ; is a major inducement of HCC. We had reported two transgenic mice models of HBsAg and HBx gene which developed highly differentiated HCCs, while HBsAg transgenic mice at 15 months old, 3 months earlier than HBx transgenic mice. To compare the different function of HBsAg and HBx gene in the development of HCC, proteomic analyses were carried out to find the proteins changed during the hepatocarcinogenesis of the HBx and HBsAg transgenic mice. We collected liver samples from wild type, p21HBsAg , p21HBsAg HBsAg, p21HBx , and p21HBx HBx mice at the ages of 6, 12 and18 months. The differently expressed proteins detected by 2-DE were identified by MALDI-TOF MS, and the data of PMF were online searched by MASCOT. 147 differential expressed proteins were found totally, 87 of which were identified by MALDI-TOF MS. 37 of the proteins that had been identified were enzymes involved in many metabolic processes, and 20 could be classified as receptors, 21 belonged to signal transduction pathways or cellular skeletal proteins, moreover 9 proteins were unnamed proteins without any report about their biological functions. The functions of differentially expressed proteins were analyzed. The expression of some proteins taking part in the process of glycolysis and metabolism of lipid were changed. The shortage of glycogen storage and deposit of fat might be associated with the enhancement of glycolysis and the over-mobilization of fat. The over-production of ROS and the inducement of oxidative stress could contribute to the development of HCC in both HBx and HBsAg transgenic mice. Our data also showed that the proteomic changes were different during the tumorigenesis between the HBsAg and HBx transgenic mice. For example, laminin receptor I was only up-regulated in the livers and tumors from the HBsAg gene knockin transgenic mice. Most importantly, an unnamed protein with unknown function was found related to the HBx induced HCC. A modified version of this protein was only found in the livers and tumors of the HBx gene knockin transgenic mice. The protein translocated into nucleus when co-transfected with HBx in SMMC-7721, a hepatoma cell line. All these data suggested that the molecular mechanisms of HCC development induced by HBsAg and HBx were different, for example, tenormin weight gain.
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COGGAN, C., HOOPER, R., PATTERSON, P., LOVELL, C. `Child falls: 5-10 years old: medical record review'. Auckland, Injury Prevention Research Centre, 28p., 2002. COGGAN, C., HOOPER, R., PATTERSON, P., LOVELL, C. `Falls in the home: a study of hospitalised injuries in preschool children'. Auckland, Injury Prevention Research Centre, 8p., 2002. COGGAN, C., BENNETT, S., HOOPER, R., LOVELL, C. `Auckland City community profile: opportunities for promoting a safety culture'. Auckland, Injury Prevention Research Centre, 157p., 2002. COGGAN, C., PATTERSON, P., HOOPER, R. `Impact evaluation of ACC Injury Prevention in Schools Programme'. Auckland, Injury Prevention Research Centre, 22p., 2001. DAVIES, E., KOZIOL-MCLAIN, J., CASEY, C., FANSLOW, J.L., HASSALL, I., CROTHERS, C. `Evaluation of Te Rito: ideas to get started'. Auckland Institute for Public Policy, Auckland University of Technology, 2002. FANSLOW, J.L. `Family violence intervention guidelines: child and partner abuse'. Wellington, Ministry of Health, 2002. HAND, J., ELIZABETH, V., BETSAN, M., RAUWHERO, H., BURTON, M., SELBY, S., FALANITULE, L. `Free from abuse: what women say and what can be done'. Auckland District Health Board, 182p., 2002. KOZIOL-MCLAIN, J., DAVIES, E., CROTHERS, C., CASEY, C., FANSLOW, J.L., HASSALL, I. `Evaluation of Te Rito: a national reporting index'. Auckland Institute for Public Policy, Auckland University of Technology, 2002 and
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